Part 6 · The patient is on therapy and something is wrong Pharmacology 55 min

Troubleshooting: Non-Response, Adverse Effects, and Difficult Conversations

The scenarios that actually fill your inbox — 'it stopped working,' acne, gynecomastia, mood changes, testicular atrophy, injection site problems, hair loss, and the patient who is buying testosterone online.

Learning objectives for this Part
  1. Evaluate the man whose symptoms have not responded to therapy despite a therapeutic testosterone level.
  2. Manage common adverse effects of testosterone therapy without unnecessary discontinuation.
  3. Counsel patients on testicular atrophy, hair loss, and acne using accurate expectations.
  4. Address non-prescribed testosterone and anabolic steroid use in a clinically productive way.

Guidelines tell you how to start therapy. They say much less about month seven, when the patient emails to say it stopped working, or month three, when his partner is asking about the acne. This Part is the troubleshooting manual.

'It Stopped Working'

Approach to apparent non-response
  1. 1
    Confirm the level was drawn at the correct time for the formulation
  2. 2
    Confirm adherence and technique — missed doses, gel washed off, wrong injection site
  3. 3
    Is the level actually at target (roughly 400–700 ng/dL)?
  4. 4
    If yes: reopen the differential — OSA, depression, thyroid, anemia, medications, relationship and situational factors
  5. 5
    If no: adjust interval before dose; verify the product and pharmacy
  6. 6
    Reassess the specific symptom that was targeted, not a global sense of wellness
The most common cause of non-response

In practice, the most common reason a man at a therapeutic testosterone level still feels poorly is that testosterone deficiency was not the primary driver of his symptoms. Untreated sleep apnea and untreated depression are the two leading culprits. Escalating the dose in this situation produces adverse effects without benefit.

Adverse Effect Playbook

ProblemLikely mechanismFirst-line management
ErythrocytosisAndrogen-driven erythropoiesis; peak-concentration dependentSee Part V thresholds. Shorten interval, reduce dose, treat OSA, consider transdermal switch, phlebotomy if needed.
Acne, oily skinAndrogen effect on sebaceous glands; often peak-relatedUsually improves after the first few months. Topical retinoid or benzoyl peroxide; reduce peak by shortening the dosing interval; dermatology if severe or scarring.
Gynecomastia or breast tendernessAromatization to estradiol, often peak-drivenReduce peaks by shortening interval or lowering dose. Measure estradiol with a sensitive assay only if it will change management. Examine for asymmetry or a discrete mass, which requires imaging.
Testicular atrophyLH suppression reduces Leydig cell stimulation; expected pharmacologyCounsel that this is expected and usually partly reversible on discontinuation. hCG can maintain testicular volume, but should be framed as fertility-directed and off-label for this indication.
Fluid retention, ankle edemaSodium and water retention; more prominent in heart failure and CKDReduce dose. Reassess cardiac and renal status. Avoid or discontinue in decompensated heart failure.
Mood lability, irritabilityOften peak-related in men on long-interval injectionsShorten the interval. Screen for underlying mood disorder. Supraphysiologic levels should be corrected downward.
Worsening sleep apnea or new snoringTestosterone can worsen OSA in susceptible menSleep study. Treat OSA. Reduce dose or hold pending evaluation if severe.
Male pattern hair lossDHT effect in genetically susceptible menCounsel before starting. Topical minoxidil; finasteride is an option but discuss its effects on sexual function and PSA interpretation.
Injection-site pain, nodulesOil vehicle, needle gauge, technique, IM depthSwitch to subcutaneous with a smaller-gauge needle, rotate sites, warm the vial, inject slowly.
Skin reaction to gel or patchVehicle irritation, alcohol contentRotate sites, try a different vehicle or brand, switch to injectable.
Elevated blood pressureClass effect noted for oral undecanoate and SC enanthate auto-injectorMonitor at every visit. Treat hypertension. Reconsider the formulation if the rise is attributable to it.
Clinical pearl

Most testosterone adverse effects are peak-concentration problems disguised as dose problems. Before you reduce the total weekly dose and lose efficacy, try delivering the same dose more often.

The Patient Buying Testosterone Online

You will encounter men using testosterone or anabolic agents obtained from online vendors, peptide sellers, or a gym. Reflexive moralizing ends the conversation and the patient continues without any medical oversight. A more productive approach is to acknowledge the goal, establish what is actually being taken and at what dose, obtain baseline safety labs including hematocrit, lipids, liver enzymes, and PSA where appropriate, and explain the specific risks that apply — erythrocytosis, hypertension, cardiomyopathy at supraphysiologic doses, infertility, and the fact that product contents are unverified.

  • Ask what, how much, from where, and for how long — without judgment.
  • Check hematocrit, lipids, comprehensive metabolic panel, PSA where age-appropriate, and blood pressure.
  • Discuss infertility explicitly, and offer semen analysis and referral if fertility matters to him.
  • Discuss what a supervised, physiologic regimen would look like if he meets criteria.
  • Document the discussion, the risks disclosed, and the plan.
  • Recognize anabolic steroid use disorder and refer when body-image pathology or dependence is present.
Case 6

At target and still tired

A 45-year-old man has been on testosterone enanthate 80 mg SC weekly for six months. His trough total testosterone is 585 ng/dL and hematocrit is 47 percent. He reports that libido and morning erections improved substantially in the first two months, but his fatigue and low mood are unchanged. He asks for a dose increase, saying he read online that he needs to be 'over 900.'

How do you respond?

Self-assessment

Check yourself before moving on

1. A man on stable testosterone therapy with a level of 560 ng/dL reports persistent fatigue despite improved libido. What is the most appropriate next step?

2. Which of the following is the most appropriate first-line approach to new gynecomastia in a man on every-2-week testosterone cypionate?

References for this Part
  1. 1.Bhasin S, Brito JP, Cunningham GR, et al. Testosterone therapy in men with hypogonadism: an Endocrine Society clinical practice guideline. J Clin Endocrinol Metab. 2018;103(5):1715–1744. Source
  2. 2.Burnett AL, Nehra A, Breau RH, et al. Erectile dysfunction: AUA guideline. J Urol. 2018;200(3):633–641. Amended 2018. Source
  3. 3.Tan RS, Cook KR, Reilly WG. Myocardial infarction and stroke risk in young healthy men treated with injectable testosterone. Int J Endocrinol. 2015;2015:970750. Source Cited only as historical context for supraphysiologic dosing claims circulating in non-clinical channels.
  4. 4.Rambhatla A, Mills JN, Rajfer J. The role of estrogen modulators in male hypogonadism and infertility. Rev Urol. 2016;18(2):66–72. Source Cited for the pharmacology of off-label aromatase inhibition, which no society guideline recommends routinely.
Full course reference list